| online with live classes, printed coursebook and virtual classroom | ||
| Start Date: 5 OCT 2025 | ||
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until October 18 (*) + 495€ for the Master's Final Project if taken |
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| 90 ECTS | ||
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12 months of free use of our nutrition software |
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ONLY AVAILABLE IN SPANISH
University-Specific Master's Degree
Clinical Nutrition and Endocrinology
The ICNS Master in Clinical Nutrition and Endocrinology has been designed by professionals who are actively engaged in clinical practice in the fields of clinical nutrition, hospital nutrition, digestive medicine and surgery, being a training program oriented towards the nutritional treatment of pathologies. During the course the most important scientific evidence related to each module is reviewed, integrating scientific aspects with clinical practice, so that the study of multiple pathologies is integrated with the experience of clinical treatment and clinical cases collected in consultation by the teachers.The program covers the main determinants of metabolic health, interpretation of analytical tests, applied diet therapy, diabetes, metabolic syndrome and insulin resistance, thyroid pathologies, autoimmunity and inflammation, cardiovascular pathologies, liver pathologies, cardiometabolic pathologies, cancer, renal pathology, urology, nutrition in surgical patients, hospital nutrition, etc. The Master includes classes entirely dedicated to clinical cases.
The Master in Clinical Nutrition and Endocrinology is one of the most complete clinically oriented programs in existence, with the recognition of training in ICNS, and the possibility of obtaining a highly recognized and valued qualification in the clinic.
Due to the fact that a significant part of the course content is updated each year, the description of each topic's points is for guidance only. The written syllabus is also complementary to the classes, where many additional points may be covered.
Professors
Dr.Álvaro CampilloMaría Fraile
Alfonso Bordallo
Dra.Isabel Barceló
Víctor Robledo
- Evidence-based education.
- Professors with extensive clinical experience.
- Clinical focus from the very first class.
- Hundreds of scientific studies are reviewed and cross-referenced throughout the course.
- Early enrollment discounts available.
- Interest-free installment payment options.
- Special group conditions for clinics.


1. WHY THE CURRENT APPROACH TO CLINICAL NUTRITION IS NEITHER NUTRITIONAL NOR CLINICAL
2. BODY COMPOSITION
2.1. BMI AS A HEALTH INDICATOR
2.2. TOTAL WEIGHT VS. BODY COMPOSITION
3. IMPORTANT CONSIDERATIONS ABOUT THE DIFFERENT MACRONUTRIENTS BEYOND DISTRIBUTION AND CALORIES
3.1. PROTEINS
3.2. FATS
3.3. CARBOHYDRATES
4. DIET AND THE ELDERLY
4.1. MOST NOTABLE PHYSIOLOGICAL CHANGES
4.2. IMPORTANT POINTS TO CONSIDER IN DIETARY HISTORY
4.3. NUTRITIONAL CONSIDERATIONS TO TAKE INTO ACCOUNT TO PREVENT FRAGILITY IN OLDER ADULTS
5. CLINICAL NUTRITION IN PRACTICE
5.1. WHAT TO DO WHEN A NEW PATIENT ARRIVES FOR A CONSULTATION?
5.2. IN-DEPTH NUTRITIONAL INTERVIEW
5.3. DRUG-FOOD-SUPPLEMENT INTERACTIONS
5.4. MEDICATIONS: ON AN EMPTY STOMACH OR WITH FOOD?
5.5. DRUG INTERACTIONS WITH FRUIT JUICES
5.6. SYNTHETIC LEVOTHYROXINE: EUTIROX®, DEXNÓN®, ETC.
5.7. ORAL ANTICOAGULANTS: VITAMIN K ANTAGONISTS: WARFARIN, ACENOCUMAROL SINTROM®
5.8. SOME INTERACTIONS BETWEEN MEDICINAL PLANTS AND SUPPLEMENTS WITH COMMONLY USED DRUGS
5.9. CAN SOME INTERACTIONS BE USED FOR THERAPEUTIC PURPOSES?
5.10. EXCIPIENTS IN MEDICINES AND SUPPLEMENTS

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SUN. 5 OCT 2025 4:00 PM to 8:00 PM
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María Fraile
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1. WHY THE CURRENT APPROACH TO CLINICAL NUTRITION IS NEITHER NUTRITIONAL NOR CLINICAL
2. BODY COMPOSITION
2.1. BMI AS A HEALTH INDICATOR
2.2. TOTAL WEIGHT VS. BODY COMPOSITION
3. IMPORTANT CONSIDERATIONS ABOUT THE DIFFERENT MACRONUTRIENTS BEYOND DISTRIBUTION AND CALORIES
3.1. PROTEINS
3.2. FATS
3.3. CARBOHYDRATES
4. DIET AND THE ELDERLY
4.1. MOST NOTABLE PHYSIOLOGICAL CHANGES
4.2. IMPORTANT POINTS TO CONSIDER IN DIETARY HISTORY
4.3. NUTRITIONAL CONSIDERATIONS TO TAKE INTO ACCOUNT TO PREVENT FRAGILITY IN OLDER ADULTS
5. CLINICAL NUTRITION IN PRACTICE
5.1. WHAT TO DO WHEN A NEW PATIENT ARRIVES FOR A CONSULTATION?
5.2. IN-DEPTH NUTRITIONAL INTERVIEW
5.3. DRUG-FOOD-SUPPLEMENT INTERACTIONS
5.4. MEDICATIONS: ON AN EMPTY STOMACH OR WITH FOOD?
5.5. DRUG INTERACTIONS WITH FRUIT JUICES
5.6. SYNTHETIC LEVOTHYROXINE: EUTIROX®, DEXNÓN®, ETC.
5.7. ORAL ANTICOAGULANTS: VITAMIN K ANTAGONISTS: WARFARIN, ACENOCUMAROL SINTROM®
5.8. SOME INTERACTIONS BETWEEN MEDICINAL PLANTS AND SUPPLEMENTS WITH COMMONLY USED DRUGS
5.9. CAN SOME INTERACTIONS BE USED FOR THERAPEUTIC PURPOSES?
5.10. EXCIPIENTS IN MEDICINES AND SUPPLEMENTS

1. BASIC PRINCIPLES
2. RED BLOOD CELLS: ANEMIC SYNDROME AND ITS DIAGNOSTIC ORIENTATION
2.1 BIOGRAPHY OF A HEMOGLOBIN OR RED BLOOD CELL (RBC)
2.1.1. TRANSFERRIN
2.2 CLARIFYING CONCEPTS
2.2.1. TYPES OF ANEMIA ACCORDING TO MCC
2.2.2. TYPES OF ANEMIA ACCORDING TO THE MEDULLARY PROLIFERATION REACTION
2.2.3. ACUTE BLEEDING VS CHRONIC BLEEDING
2.3 CONCEPT OF ANEMIA AND ITS DIFFERENTIAL DIAGNOSIS
2.4 HEMOLYSIS
3. WHITE BLOOD CELL SERIES
4. PLATELET SERIES
5. LIVER PROFILE
5.1. CONSIDERATIONS ON LIVER LESION PATTERNS
6. GLUCOSE PROFILE
7. LIPID PROFILE AND CARDIOVASCULAR RISK
8. CREATININE, CREATININE CLEARANCE, AND KIDNEY FUNCTION
9. LABORATORY CHRONOBIOLOGY
10. EFFECT OF DRUGS ON ANALYSIS
11. THE CONTROVERSY OF VITAMIN D IN ANALYSIS
12. CONSIDERATIONS ON BLOOD SEDIMENTATION RATE (BSR)

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SUN. 19 OCT 2025 4:00 PM to 8:00 PM
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Dr.Álvaro Campillo
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1. BASIC PRINCIPLES
2. RED BLOOD CELLS: ANEMIC SYNDROME AND ITS DIAGNOSTIC ORIENTATION
2.1 BIOGRAPHY OF A HEMOGLOBIN OR RED BLOOD CELL (RBC)
2.1.1. TRANSFERRIN
2.2 CLARIFYING CONCEPTS
2.2.1. TYPES OF ANEMIA ACCORDING TO MCC
2.2.2. TYPES OF ANEMIA ACCORDING TO THE MEDULLARY PROLIFERATION REACTION
2.2.3. ACUTE BLEEDING VS CHRONIC BLEEDING
2.3 CONCEPT OF ANEMIA AND ITS DIFFERENTIAL DIAGNOSIS
2.4 HEMOLYSIS
3. WHITE BLOOD CELL SERIES
4. PLATELET SERIES
5. LIVER PROFILE
5.1. CONSIDERATIONS ON LIVER LESION PATTERNS
6. GLUCOSE PROFILE
7. LIPID PROFILE AND CARDIOVASCULAR RISK
8. CREATININE, CREATININE CLEARANCE, AND KIDNEY FUNCTION
9. LABORATORY CHRONOBIOLOGY
10. EFFECT OF DRUGS ON ANALYSIS
11. THE CONTROVERSY OF VITAMIN D IN ANALYSIS
12. CONSIDERATIONS ON BLOOD SEDIMENTATION RATE (BSR)

1. SUPPLEMENTS
2. EATING, EXERCISE AND REST: KEYS AND EFFECTS
3. THE BASIS OF GOOD HEALTH IS NOT ISOLATED SUBSTANCES.
4. LEGISLATION AND CONTROL OF QUALITY, SAFETY AND INNOCUOUSNESS
5. SCIENCE AND SUPPLEMENTATION
6. CUSTOMIZING SUPPLEMENTATION
7. DIFFERENCES BETWEEN COMPOSITION TABLES AND RECOMMENDATIONS
8. INTAKE DOES NOT EQUAL ABSORPTION
8.1. PROTEIN
8.2. FATS
8.3. CARBOHYDRATES
8.4. IRON
8.5. CALCIUM
9. PROTEIN POWDER
10. ASHWAGANDHA
11. OTHER SUPPLEMENTS AGAINST STRESS AND FATIGUE
12. CAFFEINE AND COFFEE
13. VITAMIN C
14. VINEGAR, GARLIC AND CINNAMON
15. VITAMIN B3 (NIACIN)
16. VITAMIN B9
17. VITAMIN D
18. MULTIVITAMINS
19. CHROMIUM
20. MAGNESIUM
21. ZINC
22. SELENIUM
23. TURMERIC / CURCUMIN
24. GLUTAMINE, INTESTINAL PERMEABILITY AND EII
25. COMMON DEFICIENCIES AND RISKS IN EII AND DIGESTIVES
26. VITAMIN B12, VEGETARIANS AND PATIENTS WITH MALABSORPTION/DIGESTIVE DISORDERS
27. CREATINE
28. FUNCTIONS OF THE REST OF VITAMINS AND MINERALS
28.1. FAT-SOLUBLE VITAMINS
28.2. WATER-SOLUBLE VITAMINS
28.3. MINERALS
28.3.1. MACROELEMENTS
28.3.2. TRACE ELEMENTS
28.3.3. ULTRATRACE ELEMENTS
29. VITAMIN B12, INTRODUCTION AND DIETARY SOURCES
29.1. VITAMIN B12, HISTORY, DISCOVERY AND PRODUCTION
29.2. FUNCTIONS OF VITAMIN B12
29.3. PHYSIOLOGICAL AND NON-PHYSIOLOGICAL FORMS OF VITAMIN B12 29.4. DIETARY SOURCES OF VITAMIN B12
29.4. DIETARY SOURCES OF VITAMIN B12 29.5.
29.5. THE PROBLEM OF VITAMIN B12 ANALOGUES
30. BIOCHEMISTRY OF VITAMIN B12
30.1. FUNCTIONS, METABOLIC REACTIONS, HOW A DEFICIT AFFECTS AND THE FOLATE TRAP
31. VITAMIN B12 METABOLISM
31.1. METABOLISM, TRANSPORT AND STORAGE OF VITAMIN B12 31.2.
31.2. ABSORPTION AND DIFFERENCES BETWEEN PHYSIOLOGIC AND PHARMACOLOGIC DOSES
31.3. HOMOCYSTEINE AND METHYLMALONIC ACID
32. VITAMIN B12 DEFICIENCY
32.1. SPANISH VEGETARIAN POPULATION: B12 AND METHYLMALONIC ACID
32.2. OFFICIAL" RECOMMENDED DAILY ALLOWANCES AND SUPPLEMENTATION
32.3. VITAMIN B12 IN OVO-LACTO VEGETARIANS
33. SYMPTOMATOLOGY AND DIAGNOSIS OF VITAMIN B12 DEFICIENCY
33.1. VITAMIN B12 DEFICIENCY
33.2. PROGRESSIVE STAGES OF A VITAMIN B12 DEFICIENCY 33.3.
33.3. SYMPTOMATOLOGY OF VITAMIN B12 DEFICIENCY 33.4.
33.4. ANALYTICAL AND RANGE VALUES
33.4.1. METHYLMALONIC ACID
33.5. VERY HIGH VITAMIN B12
34. VITAMIN B12, PREGNANCY AND LACTATION
34.1 VITAMIN B12 IN MOTHERS AND INFANTS
35. VITAMIN B12 SUPPLEMENTATION
35.1. FORMS OF VITAMIN B12 IN SUPPLEMENTATION: CYANO, HYDROXO, METHYL
35.2. ORAL VS. PARENTERAL SUPPLEMENTATION
35.3. ADVERSE REACTIONS
35.4. REMONTE PROTOCOLS
35.5. ABSORBED AND RETAINED PERCENTAGES IN DIFFERENT DOSES AND ROUTES

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SUN. 2 NOV 2025 4:00 PM to 8:00 PM
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Víctor Robledo
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1. SUPPLEMENTS
2. EATING, EXERCISE AND REST: KEYS AND EFFECTS
3. THE BASIS OF GOOD HEALTH IS NOT ISOLATED SUBSTANCES.
4. LEGISLATION AND CONTROL OF QUALITY, SAFETY AND INNOCUOUSNESS
5. SCIENCE AND SUPPLEMENTATION
6. CUSTOMIZING SUPPLEMENTATION
7. DIFFERENCES BETWEEN COMPOSITION TABLES AND RECOMMENDATIONS
8. INTAKE DOES NOT EQUAL ABSORPTION
8.1. PROTEIN
8.2. FATS
8.3. CARBOHYDRATES
8.4. IRON
8.5. CALCIUM
9. PROTEIN POWDER
10. ASHWAGANDHA
11. OTHER SUPPLEMENTS AGAINST STRESS AND FATIGUE
12. CAFFEINE AND COFFEE
13. VITAMIN C
14. VINEGAR, GARLIC AND CINNAMON
15. VITAMIN B3 (NIACIN)
16. VITAMIN B9
17. VITAMIN D
18. MULTIVITAMINS
19. CHROMIUM
20. MAGNESIUM
21. ZINC
22. SELENIUM
23. TURMERIC / CURCUMIN
24. GLUTAMINE, INTESTINAL PERMEABILITY AND EII
25. COMMON DEFICIENCIES AND RISKS IN EII AND DIGESTIVES
26. VITAMIN B12, VEGETARIANS AND PATIENTS WITH MALABSORPTION/DIGESTIVE DISORDERS
27. CREATINE
28. FUNCTIONS OF THE REST OF VITAMINS AND MINERALS
28.1. FAT-SOLUBLE VITAMINS
28.2. WATER-SOLUBLE VITAMINS
28.3. MINERALS
28.3.1. MACROELEMENTS
28.3.2. TRACE ELEMENTS
28.3.3. ULTRATRACE ELEMENTS
29. VITAMIN B12, INTRODUCTION AND DIETARY SOURCES
29.1. VITAMIN B12, HISTORY, DISCOVERY AND PRODUCTION
29.2. FUNCTIONS OF VITAMIN B12
29.3. PHYSIOLOGICAL AND NON-PHYSIOLOGICAL FORMS OF VITAMIN B12 29.4. DIETARY SOURCES OF VITAMIN B12
29.4. DIETARY SOURCES OF VITAMIN B12 29.5.
29.5. THE PROBLEM OF VITAMIN B12 ANALOGUES
30. BIOCHEMISTRY OF VITAMIN B12
30.1. FUNCTIONS, METABOLIC REACTIONS, HOW A DEFICIT AFFECTS AND THE FOLATE TRAP
31. VITAMIN B12 METABOLISM
31.1. METABOLISM, TRANSPORT AND STORAGE OF VITAMIN B12 31.2.
31.2. ABSORPTION AND DIFFERENCES BETWEEN PHYSIOLOGIC AND PHARMACOLOGIC DOSES
31.3. HOMOCYSTEINE AND METHYLMALONIC ACID
32. VITAMIN B12 DEFICIENCY
32.1. SPANISH VEGETARIAN POPULATION: B12 AND METHYLMALONIC ACID
32.2. OFFICIAL" RECOMMENDED DAILY ALLOWANCES AND SUPPLEMENTATION
32.3. VITAMIN B12 IN OVO-LACTO VEGETARIANS
33. SYMPTOMATOLOGY AND DIAGNOSIS OF VITAMIN B12 DEFICIENCY
33.1. VITAMIN B12 DEFICIENCY
33.2. PROGRESSIVE STAGES OF A VITAMIN B12 DEFICIENCY 33.3.
33.3. SYMPTOMATOLOGY OF VITAMIN B12 DEFICIENCY 33.4.
33.4. ANALYTICAL AND RANGE VALUES
33.4.1. METHYLMALONIC ACID
33.5. VERY HIGH VITAMIN B12
34. VITAMIN B12, PREGNANCY AND LACTATION
34.1 VITAMIN B12 IN MOTHERS AND INFANTS
35. VITAMIN B12 SUPPLEMENTATION
35.1. FORMS OF VITAMIN B12 IN SUPPLEMENTATION: CYANO, HYDROXO, METHYL
35.2. ORAL VS. PARENTERAL SUPPLEMENTATION
35.3. ADVERSE REACTIONS
35.4. REMONTE PROTOCOLS
35.5. ABSORBED AND RETAINED PERCENTAGES IN DIFFERENT DOSES AND ROUTES

1. A BRIEF HISTORY OF INSULIN AND ITS RELATIONSHIP WITH DIABETES
1.1. INSULIN RESISTANCE + HYPERINSULINEMIA
2. METABOLIC SYNDROME AND ITS RELATIONSHIP WITH DIABETES AND CHRONIC METABOLIC DISEASES
2.1. WHAT IS INSULIN RESISTANCE AND HOW CAN IT BE MEASURED?
2.2. HOMA-IR, FASTING BLOOD GLUCOSE, PANCREATIC FUNCTION, AND HOW THEY ARE RELATED
3. INSULIN RESISTANCE, NON-ENZYMATIC GLYCOSYLATION COMPOUNDS, AND METABOLIC SYNDROME
3.1. LIPOLYTIC METABOLISM AND KETONE BODIES
4. ADIPOSE TISSUE, INSULIN RESISTANCE, AND METABOLIC SYNDROME
4.1. ENDOCRINE CHANGES ASSOCIATED WITH OBESITY
4.1.1. ENDOCRINE PANCREAS
4.1.2. ADIPOCYTES, EXCESS AND DEFICIENCY
5. ARTERIAL HYPERTENSION, INSULIN RESISTANCE, OBESITY, AND THINNESS
6. NUTRITIONAL AND LIFESTYLE CONSIDERATIONS IN SDMET

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SUN. 16 NOV 2025 4:00 PM to 8:00 PM
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Dr.Álvaro Campillo
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1. A BRIEF HISTORY OF INSULIN AND ITS RELATIONSHIP WITH DIABETES
1.1. INSULIN RESISTANCE + HYPERINSULINEMIA
2. METABOLIC SYNDROME AND ITS RELATIONSHIP WITH DIABETES AND CHRONIC METABOLIC DISEASES
2.1. WHAT IS INSULIN RESISTANCE AND HOW CAN IT BE MEASURED?
2.2. HOMA-IR, FASTING BLOOD GLUCOSE, PANCREATIC FUNCTION, AND HOW THEY ARE RELATED
3. INSULIN RESISTANCE, NON-ENZYMATIC GLYCOSYLATION COMPOUNDS, AND METABOLIC SYNDROME
3.1. LIPOLYTIC METABOLISM AND KETONE BODIES
4. ADIPOSE TISSUE, INSULIN RESISTANCE, AND METABOLIC SYNDROME
4.1. ENDOCRINE CHANGES ASSOCIATED WITH OBESITY
4.1.1. ENDOCRINE PANCREAS
4.1.2. ADIPOCYTES, EXCESS AND DEFICIENCY
5. ARTERIAL HYPERTENSION, INSULIN RESISTANCE, OBESITY, AND THINNESS
6. NUTRITIONAL AND LIFESTYLE CONSIDERATIONS IN SDMET

1. INTERMITTENT FASTING
1.1. BRIEF PHYSIOPATHOLOGICAL OVERVIEW OF FASTING
1.2. TYPES OF INTERMITTENT FASTING
1.3. INTERMITTENT FASTING AND CANCER
1.4. INTERMITTENT FASTING AND SOCIAL JET LAG (CRONODISRUPCIÓN)
1.5. INTERMITTENT FASTING AND METABOLIC SYNDROME
1.6. INTERMITTENT FASTING AND DIABETIC KIDNEY DISEASE
1.7. INTERMITTENT FASTING AND AUTOIMMUNE DISEASES
1.8. INTERMITTENT FASTING AND PREMATURE AGING (INFLAMMING)
2. THE MEDITERRANEAN DIET
2.1. NUMEROUS DEFINITIONS OF THE MEDITERRANEAN DIET AND A COMMON MODEL
2.2. THE DIOGENES PROJECT AS A SCIENTIFIC EVOLUTION OF THE MEDITERRANEAN DIET
2.3. GLYCEMIC INDEX VERSUS GLYCEMIC LOAD
2.4. CLINICAL BENEFITS OF THE MEDITERRANEAN DIET
3. LIPOLYTIC DIETS: KETOGENIC AND LOW-CARB
3.1. GLYCOLYTIC METABOLISM VERSUS LIPOLYTIC METABOLISM
3.2. NUTRITIONAL KETOSIS VERSUS KETOACIDOSIS
3.3. ORIGINS OF KETOGENIC DIETS
3.4. LIPOLYTIC DIETS: LOW-CARB DIETS AND KETOGENIC DIETS
3.5. STANDARD KETOGENIC DIET, THERAPEUTIC KETOGENIC DIET AND STANDARD BALANCED DIET
3.6. SPANISH MEDITERRANEAN KETOGENIC DIET
3.7. KETOADAPTATION: OPTIMIZED LIPOLYTIC METABOLISM
3.8. URINE TEST STRIPS, KETOSIS, AND KETOADAPTATION
3.9. KETO FLU OR KETO FLU
3.10. THERAPEUTIC USES OF LIPOLYTIC DIETS
4. DIETARY STRATEGIES AND CHARACTERISTICS OF PREMATURE AGING

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SUN. 7 DIC 2025 4:00 PM to 8:00 PM
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Dr.Álvaro Campillo
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1. INTERMITTENT FASTING
1.1. BRIEF PHYSIOPATHOLOGICAL OVERVIEW OF FASTING
1.2. TYPES OF INTERMITTENT FASTING
1.3. INTERMITTENT FASTING AND CANCER
1.4. INTERMITTENT FASTING AND SOCIAL JET LAG (CRONODISRUPCIÓN)
1.5. INTERMITTENT FASTING AND METABOLIC SYNDROME
1.6. INTERMITTENT FASTING AND DIABETIC KIDNEY DISEASE
1.7. INTERMITTENT FASTING AND AUTOIMMUNE DISEASES
1.8. INTERMITTENT FASTING AND PREMATURE AGING (INFLAMMING)
2. THE MEDITERRANEAN DIET
2.1. NUMEROUS DEFINITIONS OF THE MEDITERRANEAN DIET AND A COMMON MODEL
2.2. THE DIOGENES PROJECT AS A SCIENTIFIC EVOLUTION OF THE MEDITERRANEAN DIET
2.3. GLYCEMIC INDEX VERSUS GLYCEMIC LOAD
2.4. CLINICAL BENEFITS OF THE MEDITERRANEAN DIET
3. LIPOLYTIC DIETS: KETOGENIC AND LOW-CARB
3.1. GLYCOLYTIC METABOLISM VERSUS LIPOLYTIC METABOLISM
3.2. NUTRITIONAL KETOSIS VERSUS KETOACIDOSIS
3.3. ORIGINS OF KETOGENIC DIETS
3.4. LIPOLYTIC DIETS: LOW-CARB DIETS AND KETOGENIC DIETS
3.5. STANDARD KETOGENIC DIET, THERAPEUTIC KETOGENIC DIET AND STANDARD BALANCED DIET
3.6. SPANISH MEDITERRANEAN KETOGENIC DIET
3.7. KETOADAPTATION: OPTIMIZED LIPOLYTIC METABOLISM
3.8. URINE TEST STRIPS, KETOSIS, AND KETOADAPTATION
3.9. KETO FLU OR KETO FLU
3.10. THERAPEUTIC USES OF LIPOLYTIC DIETS
4. DIETARY STRATEGIES AND CHARACTERISTICS OF PREMATURE AGING


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SUN. 21 DIC 2025 4:00 PM to 8:00 PM
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María Fraile
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2. CLINICAL CASES OF METABOLIC PATHOLOGIES: INSULIN RESISTANCE, DM2, METABOLIC SYNDROME, HEPATIC STEATOSIS.

1. NUTRITIONAL EPIDEMIOLOGY
1.2. NUTRITION AND PUBLIC HEALTH
2. CARDIOVASCULAR RISK
2.1. CHOLESTEROL
2.2. CLINICAL RATIONALE AND MARKERS
3. NUTRITIONAL MYTHS
3.1. OUNCES AND HEALTH
3.2. RED WINE AND HEALTH
3.3. GRAINS AND HEALTH
3.4. MILK PRODUCTS
3.5. POISSON AND OMEGA 3
3.6. GLUCIDES, GLUCOSE AND HEALTH
3.7. GLYCEMIC INDEX
3.8. COMPLETE FOODS
3.9. VÉGÉTALISME
3.10. INTERMITTENT JEÛNE
3.11. SWEETENERS
3.12. ANTIOXYDANTS
3.13. COMPLÉMENTS
3.14. DIÉTÉTOTHÉRAPIE QUANTITATIVE
3.15. WEIGHT LOSS AS A CALORIC ISSUE
3.16. UAE
4. FOOD AND PESTICIDES
5. THE TRANSFORMATION OF MENTAL HEALTH INTO A NUTRITIONAL PROBLEM
6. CONCLUSION

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SUN. 11 ENE 2026 4:00 PM to 8:00 PM
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Alfonso Bordallo
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1. NUTRITIONAL EPIDEMIOLOGY
1.2. NUTRITION AND PUBLIC HEALTH
2. CARDIOVASCULAR RISK
2.1. CHOLESTEROL
2.2. CLINICAL RATIONALE AND MARKERS
3. NUTRITIONAL MYTHS
3.1. OUNCES AND HEALTH
3.2. RED WINE AND HEALTH
3.3. GRAINS AND HEALTH
3.4. MILK PRODUCTS
3.5. POISSON AND OMEGA 3
3.6. GLUCIDES, GLUCOSE AND HEALTH
3.7. GLYCEMIC INDEX
3.8. COMPLETE FOODS
3.9. VÉGÉTALISME
3.10. INTERMITTENT JEÛNE
3.11. SWEETENERS
3.12. ANTIOXYDANTS
3.13. COMPLÉMENTS
3.14. DIÉTÉTOTHÉRAPIE QUANTITATIVE
3.15. WEIGHT LOSS AS A CALORIC ISSUE
3.16. UAE
4. FOOD AND PESTICIDES
5. THE TRANSFORMATION OF MENTAL HEALTH INTO A NUTRITIONAL PROBLEM
6. CONCLUSION




1. INTRODUCTION
2. THE POWER OF HORMONES AND THE ENDOCRINE SYSTEM
2.1. WHAT ARE HORMONES?
2.2. ENDOCRINE, PARACRINE AND AUTOCRINE MECHANISM. HORMONE-RECEPTOR SPECIFICITY (H-R)
2.3. HOW ARE HORMONES CLASSIFIED?
2.4. PATHOPHYSIOLOGY OF THE MAIN ENDOCRINE SYNDROMES
2.4.1. VARIATIONS IN THE THEME OF GLANDS
2.4.2. ALTERATION IN THEIR HORMONAL ACTIVITY
3. BASIC INTERPRETATION OF THE MAIN HORMONAL STUDIES: THE HYPOTHALAMUS-PITUITARY-PITUITARY-TARGET-ORGAN AXIS
4. OTHER PHYSIOLOGICAL SYSTEMS INVOLVED IN MAINTAINING AN ADEQUATE ENDOCRINE SYSTEM AND VICE VERSA.
4.1. ADEQUATE CIRCADIAN RHYTHM
4.2. HEALTHY ADIPOSE TISSUE
4.3. PROPER MICROBIOTA AND GASTROINTESTINAL HORMONES 4.3.1.
4.3.1. CHRONOBIOLOGY OF INTESTINAL MICROBIOTA
ESTROGENS, MICROBIOTA, GASTROINTESTINAL HORMONES, DIGESTIVE AND SYSTEMIC HEALTH 4.3.3.
4.3.3. GHRELIN
4.3.4. PEPTIDE YY
4.3.5. PEPTIDE PP
4.3.6. GLP1
4.3.7. OXM
4.3.8. AMYLIN
4.5. MITOHORMESIS

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SUN. 25 ENE 2026 4:00 PM to 8:00 PM
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Dr.Álvaro Campillo
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1. INTRODUCTION
2. THE POWER OF HORMONES AND THE ENDOCRINE SYSTEM
2.1. WHAT ARE HORMONES?
2.2. ENDOCRINE, PARACRINE AND AUTOCRINE MECHANISM. HORMONE-RECEPTOR SPECIFICITY (H-R)
2.3. HOW ARE HORMONES CLASSIFIED?
2.4. PATHOPHYSIOLOGY OF THE MAIN ENDOCRINE SYNDROMES
2.4.1. VARIATIONS IN THE THEME OF GLANDS
2.4.2. ALTERATION IN THEIR HORMONAL ACTIVITY
3. BASIC INTERPRETATION OF THE MAIN HORMONAL STUDIES: THE HYPOTHALAMUS-PITUITARY-PITUITARY-TARGET-ORGAN AXIS
4. OTHER PHYSIOLOGICAL SYSTEMS INVOLVED IN MAINTAINING AN ADEQUATE ENDOCRINE SYSTEM AND VICE VERSA.
4.1. ADEQUATE CIRCADIAN RHYTHM
4.2. HEALTHY ADIPOSE TISSUE
4.3. PROPER MICROBIOTA AND GASTROINTESTINAL HORMONES 4.3.1.
4.3.1. CHRONOBIOLOGY OF INTESTINAL MICROBIOTA
ESTROGENS, MICROBIOTA, GASTROINTESTINAL HORMONES, DIGESTIVE AND SYSTEMIC HEALTH 4.3.3.
4.3.3. GHRELIN
4.3.4. PEPTIDE YY
4.3.5. PEPTIDE PP
4.3.6. GLP1
4.3.7. OXM
4.3.8. AMYLIN
4.5. MITOHORMESIS

1. THE ORIGIN OF ECV
1.1. PHYSIOPATHOLOGICAL ORIGIN OF CVD
1.2. WHAT IS INSULIN RESISTANCE AND HOW CAN IT BE MEASURED? HOW CAN AN INCREASED CARDIOVASCULAR RISK BE MEASURED?
1.3. SOCIO-CULTURAL ORIGIN OF CVD
1.4. SENATOR MCGOVERN'S DIETARY GUIDELINES
1.5. THE ROLE OF THE USDA AND THE NIH
2. ARTERIAL HYPERTENSION, ISCHEMIC HEART DISEASE, AND CVD
2.1 ARTERIAL HYPERTENSION (AHT)
2.1.1. TOBACCO
2.1.2. OBESITY
2.1.3. RELATIONSHIP BETWEEN HTN AND OVERWEIGHT AND OBESITY
2.1.4. ADVANCED AGE
2.1.5. HEART
2.1.6. PERIPHERAL VASCULAR SYSTEM
2.1.7. KIDNEY
2.1.8. RETINA
2.1.9. ROLE OF SALT IN HYPERTENSION
2.2 ISCHEMIC HEART DISEASE (IHD)
2.2.1. SYMPTOMS THAT A PATIENT WITH ACUTE MYOCARDIAL ISCHEMIA MAY EXPERIENCE
2.3. DISAPPEARING MISCONCEPTIONS ABOUT CVD
2.4 IMPORTANCE AND RISK FACTORS OF ISCHEMIC HEART DISEASE
3. NUTRITIONAL AND LIFESTYLE CONSIDERATIONS IN CVD
3.1. HOW CAN WE REDUCE INSULIN RESISTANCE?
3.2. HOW CAN WE REDUCE STRESS?
3.3. FOODS RICH IN PHYTOCHEMICALS AND BIOACTIVE SUBSTANCES WITH POTENTIAL HEALTH BENEFITS IN THE CONTEXT OF A HEALTHY LIFESTYLE
3.3.1. HOW TO PREPARE THEM

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SUN. 8 FEB 2026 4:00 PM to 8:00 PM
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|
Dr.Álvaro Campillo
|
1. THE ORIGIN OF ECV
1.1. PHYSIOPATHOLOGICAL ORIGIN OF CVD
1.2. WHAT IS INSULIN RESISTANCE AND HOW CAN IT BE MEASURED? HOW CAN AN INCREASED CARDIOVASCULAR RISK BE MEASURED?
1.3. SOCIO-CULTURAL ORIGIN OF CVD
1.4. SENATOR MCGOVERN'S DIETARY GUIDELINES
1.5. THE ROLE OF THE USDA AND THE NIH
2. ARTERIAL HYPERTENSION, ISCHEMIC HEART DISEASE, AND CVD
2.1 ARTERIAL HYPERTENSION (AHT)
2.1.1. TOBACCO
2.1.2. OBESITY
2.1.3. RELATIONSHIP BETWEEN HTN AND OVERWEIGHT AND OBESITY
2.1.4. ADVANCED AGE
2.1.5. HEART
2.1.6. PERIPHERAL VASCULAR SYSTEM
2.1.7. KIDNEY
2.1.8. RETINA
2.1.9. ROLE OF SALT IN HYPERTENSION
2.2 ISCHEMIC HEART DISEASE (IHD)
2.2.1. SYMPTOMS THAT A PATIENT WITH ACUTE MYOCARDIAL ISCHEMIA MAY EXPERIENCE
2.3. DISAPPEARING MISCONCEPTIONS ABOUT CVD
2.4 IMPORTANCE AND RISK FACTORS OF ISCHEMIC HEART DISEASE
3. NUTRITIONAL AND LIFESTYLE CONSIDERATIONS IN CVD
3.1. HOW CAN WE REDUCE INSULIN RESISTANCE?
3.2. HOW CAN WE REDUCE STRESS?
3.3. FOODS RICH IN PHYTOCHEMICALS AND BIOACTIVE SUBSTANCES WITH POTENTIAL HEALTH BENEFITS IN THE CONTEXT OF A HEALTHY LIFESTYLE
3.3.1. HOW TO PREPARE THEM

1. BASIC CONCEPTS OF THE IMMUNE SYSTEM
2. MODIFIABLE RISK FACTORS IN AUTOIMMUNE DISEASES: POSSIBLE TRIGGERS
2.1. INTESTINAL HEALTH. MUCOSAL STATUS. EXCESSIVE INTESTINAL PERMEABILITY.
2.2. INFECTIONS
2.3. DRUGS. VACCINES
2.4. IMPLANTS
3. WHAT ARE THE COMMON PROBLEMS WE ENCOUNTER IN CONSULTATION WITH PATIENTS WITH AUTOIMMUNE PATHOLOGIES?
4. HOW WILL WE APPROACH MAINLY ANY AUTOIMMUNE CASE FROM OUR CLINICAL NUTRITION PRACTICE?
4.1. PHARMACOLOGICAL TREATMENT AND NUTRITION
5. NUTRITIONAL CONSIDERATIONS FOR THE APPROACH TO AIDS
5.1. NUTRIENTS AND IMMUNE FUNCTION.
5.2. FUNDAMENTAL PILLARS IN TERMS OF NUTRITION IN PEOPLE WITH AUTOIMMUNITY.
5.3. FOODS RICH IN POLYPHENOLS
5.4. FOODS AS MEDICINE
5.5. RED VEGETABLES
5.6. MANGO
5.7. CORIANDER
5.8. BRASSICACEAE OR CRUCIFEROUS VEGETABLES
5.9. WHICH FOODS AND/OR PRODUCTS ARE WORSE FOR AUTOIMMUNE DISEASES?
5.10. ADDITIVES OF COMMON USE AND MICROBIOTA 5.11. IDEAS AND EXAMPLE OF THE BEST WAY TO AVOID AUTOIMMUNE DISEASES
5.11. IDEAS AND EXAMPLES OF FOODS
SUPPLEMENTATION AND PHYTOTHERAPY IN THE MAIN AUTOIMMUNE DISORDERS 7.
7. WHAT SHOULD BE TAKEN INTO ACCOUNT BESIDES FOOD?

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SUN. 22 FEB 2026 4:00 PM to 8:00 PM
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María Fraile
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1. BASIC CONCEPTS OF THE IMMUNE SYSTEM
2. MODIFIABLE RISK FACTORS IN AUTOIMMUNE DISEASES: POSSIBLE TRIGGERS
2.1. INTESTINAL HEALTH. MUCOSAL STATUS. EXCESSIVE INTESTINAL PERMEABILITY.
2.2. INFECTIONS
2.3. DRUGS. VACCINES
2.4. IMPLANTS
3. WHAT ARE THE COMMON PROBLEMS WE ENCOUNTER IN CONSULTATION WITH PATIENTS WITH AUTOIMMUNE PATHOLOGIES?
4. HOW WILL WE APPROACH MAINLY ANY AUTOIMMUNE CASE FROM OUR CLINICAL NUTRITION PRACTICE?
4.1. PHARMACOLOGICAL TREATMENT AND NUTRITION
5. NUTRITIONAL CONSIDERATIONS FOR THE APPROACH TO AIDS
5.1. NUTRIENTS AND IMMUNE FUNCTION.
5.2. FUNDAMENTAL PILLARS IN TERMS OF NUTRITION IN PEOPLE WITH AUTOIMMUNITY.
5.3. FOODS RICH IN POLYPHENOLS
5.4. FOODS AS MEDICINE
5.5. RED VEGETABLES
5.6. MANGO
5.7. CORIANDER
5.8. BRASSICACEAE OR CRUCIFEROUS VEGETABLES
5.9. WHICH FOODS AND/OR PRODUCTS ARE WORSE FOR AUTOIMMUNE DISEASES?
5.10. ADDITIVES OF COMMON USE AND MICROBIOTA 5.11. IDEAS AND EXAMPLE OF THE BEST WAY TO AVOID AUTOIMMUNE DISEASES
5.11. IDEAS AND EXAMPLES OF FOODS
SUPPLEMENTATION AND PHYTOTHERAPY IN THE MAIN AUTOIMMUNE DISORDERS 7.
7. WHAT SHOULD BE TAKEN INTO ACCOUNT BESIDES FOOD?

1. HYPOTHYROIDISM: HASHIMOTO HYPOTHYROIDISM
1.1. EPIGENETIC FACTORS
1.2. MOST IMPORTANT ENVIRONMENTAL TRIGGERING FACTORS
1.2.1. IODINE
1.2.2. SELENIUM
1.2.3. VITAMIN D
1.2.4. DRUGS AND XENOBIOTICS
1.2.5. MICROORGANISMS
1.2.6. EARLY EXPOSURE
1.3. PATHOGENESIS
1.4. DIAGNOSIS AND ITS RELATION TO SYMPTOMATOLOGY 1.5.
SYMPTOMATOLOGY AND ASSOCIATED COMORBIDITIES 1.6.
1.6. PHARMACOLOGICAL TREATMENT
1.7. DIET THERAPY
1.8. SUPPLEMENTATION
1.9. BASIC NON-NUTRITIONAL PILLARS
2. HYPERTHYROIDISM: SEVERE DISEASE
2.1. COMORBIDITIES OR COMPLICATIONS: THYROID EYE DISEASE - GRAVID ORBITOPATHY
2.2. MICROBIOTA AND GRAVE'S DISEASE
2.3. KEY PILLARS IN THE NUTRITIONAL APPROACH TO SEVERE DISEASE
2.4. SPECIAL LIFESTYLE, DIETARY AND SUPPLEMENTATION CONSIDERATIONS IN GRAVE'S DISEASE AND GRAVE'S ORBITOPATHY. PATHOLOGIES RELATED TO THE ADRENAL GLANDS
3. ADDISON'S DISEASE
3.1. ETIOPATHOGENESIS
3.2. CLINICAL MANIFESTATIONS
3.3. AUTOIMMUNE ADDISON'S DISEASE
3.4. DIAGNOSIS AND TREATMENT
3.5. NUTRITIONAL APPROACH AND LIFESTYLE
3.6. IMPORTANT NON-NUTRITIONAL FACTORS
4. CUSHING'S SYNDROME AND DISEASE
4.1. SIGNS AND SYMPTOMS
4.2. FUNDAMENTAL NUTRITIONAL AND LIFESTYLE PILLARS TO BE TAKEN INTO ACCOUNT

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SUN. 8 MAR 2026 4:00 PM to 8:00 PM
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María Fraile
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1. HYPOTHYROIDISM: HASHIMOTO HYPOTHYROIDISM
1.1. EPIGENETIC FACTORS
1.2. MOST IMPORTANT ENVIRONMENTAL TRIGGERING FACTORS
1.2.1. IODINE
1.2.2. SELENIUM
1.2.3. VITAMIN D
1.2.4. DRUGS AND XENOBIOTICS
1.2.5. MICROORGANISMS
1.2.6. EARLY EXPOSURE
1.3. PATHOGENESIS
1.4. DIAGNOSIS AND ITS RELATION TO SYMPTOMATOLOGY 1.5.
SYMPTOMATOLOGY AND ASSOCIATED COMORBIDITIES 1.6.
1.6. PHARMACOLOGICAL TREATMENT
1.7. DIET THERAPY
1.8. SUPPLEMENTATION
1.9. BASIC NON-NUTRITIONAL PILLARS
2. HYPERTHYROIDISM: SEVERE DISEASE
2.1. COMORBIDITIES OR COMPLICATIONS: THYROID EYE DISEASE - GRAVID ORBITOPATHY
2.2. MICROBIOTA AND GRAVE'S DISEASE
2.3. KEY PILLARS IN THE NUTRITIONAL APPROACH TO SEVERE DISEASE
2.4. SPECIAL LIFESTYLE, DIETARY AND SUPPLEMENTATION CONSIDERATIONS IN GRAVE'S DISEASE AND GRAVE'S ORBITOPATHY. PATHOLOGIES RELATED TO THE ADRENAL GLANDS
3. ADDISON'S DISEASE
3.1. ETIOPATHOGENESIS
3.2. CLINICAL MANIFESTATIONS
3.3. AUTOIMMUNE ADDISON'S DISEASE
3.4. DIAGNOSIS AND TREATMENT
3.5. NUTRITIONAL APPROACH AND LIFESTYLE
3.6. IMPORTANT NON-NUTRITIONAL FACTORS
4. CUSHING'S SYNDROME AND DISEASE
4.1. SIGNS AND SYMPTOMS
4.2. FUNDAMENTAL NUTRITIONAL AND LIFESTYLE PILLARS TO BE TAKEN INTO ACCOUNT

1. OVERFED, BUT UNDERNOURISHED: THEIR RELATIONSHIP WITH CARDIOMETABOLIC PATHOLOGIES.
2. MAIN ORGANS INVOLVED IN CARDIOMETABOLIC HEALTH: MICROBIOTA AND INTESTINE, ADIPOSE TISSUE, SKELETAL MUSCLE, AND LIVER.
2.1. MICROBIOTA, INTESTINAL HEALTH AND MESENTERY
2.1.1. PERINATAL FACTORS
2.1.2. POSTNATAL FACTORS 2.2.
2.2. BREAST MILK AND MICROBIOTA 2.3.
2.3. CHRONOBIOLOGY OF THE INTESTINAL MICROBIOTA 2.4.
2.4. DISBIOSIS
2.5. MICROBIOTA AND COVID-19
2.5.1. GASTRIC ACID SECRETION
2.5.2. BACTERICIDAL EFFECT OF PANCREATIC AND BILIARY SECRETION 2.5.3.
2.5.3. INTERDIGESTIVE MIGRATORY MOTOR COMPLEXES
2.5.4. COMPETENT ILEO-CAECAL VALVE
2.6. THE MESENTERIC ORGAN
3. ADIPOSE TISSUE AS AN ENDOCRINE ORGAN
3.1. HYPERPLASIA, HYPERTROPHY AND PYROPTOSIS OF ADIPOSE TISSUE
3.2. BASAL LIPOLYSIS VS. STIMULATED LIPOLYSIS
3.3. THE 4 PHENOTYPES OF ADIPOSE TISSUE AND THEIR RELATIONSHIP TO METABOLIC HEALTH
3.3.1. METABOLIC HEALTHY NORMAL WEIGHT PHENOTYPE
3.3.2. PHENOTYPE METABOLIC UNHEALTHY NORMAL WEIGHT
3.3.3. PHENOTYPE METABOLIC HEALTHY OVERWEIGHT
3.3.4. METABOLIC UNHEALTHY UNHEALTHY OVERWEIGHT PHENOTYPE
4. SKELETAL MUSCLE AS AN ENDOCRINE ORGAN
4.1. HEART AND BLOOD VESSELS
4.2. MITOCHONDRIA
4.3. MICROBIOTA AND GUT
4.4. IMMUNE SYSTEM
4.5. LIVER
4.6. ADIPOSE TISSUE
4.7. HIGH INTENSITY AEROBIC EXERCISE
4.8. STRENGTH EXERCISE
4.9. HIGH-INTENSITY INTERVALLIC EXERCISE (HIIT)
5. LIVER, HEPATOKINES AND CARDIOMETABOLIC HEALTH
5.1. FGF21
5.2. FOLLISTATIN
5.3. HSP72
5.4. IGFBP1
5.5. SELENOPROTEIN
5.6. ANGPTL4
6. MAIN CARDIOMETABOLIC PATHOLOGIES
6.1. OBESITY, INFLAMMAGING AND PREMATURE DEATH
6.1.1. GENOMIC INSTABILITY
6.1.2. TELOMERE SHORTENING
6.1.3. EPIGENETIC ALTERATIONS
6.1.4. LOSS OF PROTEOSTASIS (AUTOPHAGY)
6.1.5. INADEQUATE NUTRIENT UPTAKE
6.1.6. CELLULAR SENESCENCE
6.1.7. MITOCHONDRIAL DYSFUNCTION
6.1.8. INTEGRATIVE FACTORS OF AGING 6.2.
6.2. CARDIOMETABOLIC HEALTH, SEDENTARY LIFESTYLE AND REDUCED FUNCTIONAL MOBILITY
6.3. CARDIOMETABOLIC HEALTH, SARCOPENIA AND METABAGING CYCLE 6.4.
6.4. GOUT AND CARDIOMETABOLIC PATHOLOGIES
6.4.1. ALCOHOL
6.4.2. SUGARY DRINKS AND FRUCTOSE-RICH PRODUCTS 6.4.3.
6.4.3. PURINES AND PROTEINS
6.5. CARDIOMETABOLIC HEALTH AND PANCREATIC PATHOLOGY 6.6.
6.6. CARDIOMETABOLIC HEALTH AND INFERTILITY IN MEN
7. TAKE-HOME LIFESTYLE TIPS ON CARDIOMETABOLIC HEALTH
8. CARDIOMETABOLIC HEALTH AND WOMEN
8.1. OBESITY AND INFERTILITY IN WOMEN
8.1.1. ANOVULATION
8.1.2. HYPERANDROGENISM
8.1.3. HYPERINSULINEMIA
8.2. LIFESTYLE TIPS CARDIOMETABOLIC HEALTH AND SOP
8.2.1. LIFESTYLE
8.2.2. MITOCHONDRIAL FUNCTION
8.2.3. METABOLIC SYNDROME AND INSULIN RESISTANCE 8.2.4.
8.2.4. VITAMIN B8
8.2.5. OMEGA-3 FATTY ACIDS
8.2.6. COENZYME Q10
8.2.7. VITAMIN D3
8.3. MENOPAUSE AND CARDIOMETABOLIC RISK
8.4. MENOPAUSE, HORMONE REPLACEMENT THERAPY AND CARDIOMETABOLIC RISK
8.4.1. WINDOW OF OPPORTUNITY
8.4.2. WOMAN'S AGE
8.4.3. CARDIOMETABOLIC RISK ASSESSMENT 8.4.4.
8.4.4. ROUTE OF ADMINISTRATION
8.4.5. THS WITH ESTROGENS VS. COMBINATION OF ESTROGENS AND PROGESTOGENS
8.5. LIFESTYLE TIPS FOR CARDIOMETABOLIC RISK IN MENOPAUSE
8.5.1. EVALUATION OF THE CHRONOTYPE OF EACH WOMAN
8.5.2. COMBINATION OF AEROBIC, STRENGTH AND PELVIC FLOOR EXERCISE
8.5.3. SUPPLEMENTS, MICRONUTRIENTS, AND BENEFICIAL SUBSTANCES IN MENOPAUSE

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SUN. 22 MAR 2026 4:00 PM to 8:00 PM
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Dr.Álvaro Campillo
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1. OVERFED, BUT UNDERNOURISHED: THEIR RELATIONSHIP WITH CARDIOMETABOLIC PATHOLOGIES.
2. MAIN ORGANS INVOLVED IN CARDIOMETABOLIC HEALTH: MICROBIOTA AND INTESTINE, ADIPOSE TISSUE, SKELETAL MUSCLE, AND LIVER.
2.1. MICROBIOTA, INTESTINAL HEALTH AND MESENTERY
2.1.1. PERINATAL FACTORS
2.1.2. POSTNATAL FACTORS 2.2.
2.2. BREAST MILK AND MICROBIOTA 2.3.
2.3. CHRONOBIOLOGY OF THE INTESTINAL MICROBIOTA 2.4.
2.4. DISBIOSIS
2.5. MICROBIOTA AND COVID-19
2.5.1. GASTRIC ACID SECRETION
2.5.2. BACTERICIDAL EFFECT OF PANCREATIC AND BILIARY SECRETION 2.5.3.
2.5.3. INTERDIGESTIVE MIGRATORY MOTOR COMPLEXES
2.5.4. COMPETENT ILEO-CAECAL VALVE
2.6. THE MESENTERIC ORGAN
3. ADIPOSE TISSUE AS AN ENDOCRINE ORGAN
3.1. HYPERPLASIA, HYPERTROPHY AND PYROPTOSIS OF ADIPOSE TISSUE
3.2. BASAL LIPOLYSIS VS. STIMULATED LIPOLYSIS
3.3. THE 4 PHENOTYPES OF ADIPOSE TISSUE AND THEIR RELATIONSHIP TO METABOLIC HEALTH
3.3.1. METABOLIC HEALTHY NORMAL WEIGHT PHENOTYPE
3.3.2. PHENOTYPE METABOLIC UNHEALTHY NORMAL WEIGHT
3.3.3. PHENOTYPE METABOLIC HEALTHY OVERWEIGHT
3.3.4. METABOLIC UNHEALTHY UNHEALTHY OVERWEIGHT PHENOTYPE
4. SKELETAL MUSCLE AS AN ENDOCRINE ORGAN
4.1. HEART AND BLOOD VESSELS
4.2. MITOCHONDRIA
4.3. MICROBIOTA AND GUT
4.4. IMMUNE SYSTEM
4.5. LIVER
4.6. ADIPOSE TISSUE
4.7. HIGH INTENSITY AEROBIC EXERCISE
4.8. STRENGTH EXERCISE
4.9. HIGH-INTENSITY INTERVALLIC EXERCISE (HIIT)
5. LIVER, HEPATOKINES AND CARDIOMETABOLIC HEALTH
5.1. FGF21
5.2. FOLLISTATIN
5.3. HSP72
5.4. IGFBP1
5.5. SELENOPROTEIN
5.6. ANGPTL4
6. MAIN CARDIOMETABOLIC PATHOLOGIES
6.1. OBESITY, INFLAMMAGING AND PREMATURE DEATH
6.1.1. GENOMIC INSTABILITY
6.1.2. TELOMERE SHORTENING
6.1.3. EPIGENETIC ALTERATIONS
6.1.4. LOSS OF PROTEOSTASIS (AUTOPHAGY)
6.1.5. INADEQUATE NUTRIENT UPTAKE
6.1.6. CELLULAR SENESCENCE
6.1.7. MITOCHONDRIAL DYSFUNCTION
6.1.8. INTEGRATIVE FACTORS OF AGING 6.2.
6.2. CARDIOMETABOLIC HEALTH, SEDENTARY LIFESTYLE AND REDUCED FUNCTIONAL MOBILITY
6.3. CARDIOMETABOLIC HEALTH, SARCOPENIA AND METABAGING CYCLE 6.4.
6.4. GOUT AND CARDIOMETABOLIC PATHOLOGIES
6.4.1. ALCOHOL
6.4.2. SUGARY DRINKS AND FRUCTOSE-RICH PRODUCTS 6.4.3.
6.4.3. PURINES AND PROTEINS
6.5. CARDIOMETABOLIC HEALTH AND PANCREATIC PATHOLOGY 6.6.
6.6. CARDIOMETABOLIC HEALTH AND INFERTILITY IN MEN
7. TAKE-HOME LIFESTYLE TIPS ON CARDIOMETABOLIC HEALTH
8. CARDIOMETABOLIC HEALTH AND WOMEN
8.1. OBESITY AND INFERTILITY IN WOMEN
8.1.1. ANOVULATION
8.1.2. HYPERANDROGENISM
8.1.3. HYPERINSULINEMIA
8.2. LIFESTYLE TIPS CARDIOMETABOLIC HEALTH AND SOP
8.2.1. LIFESTYLE
8.2.2. MITOCHONDRIAL FUNCTION
8.2.3. METABOLIC SYNDROME AND INSULIN RESISTANCE 8.2.4.
8.2.4. VITAMIN B8
8.2.5. OMEGA-3 FATTY ACIDS
8.2.6. COENZYME Q10
8.2.7. VITAMIN D3
8.3. MENOPAUSE AND CARDIOMETABOLIC RISK
8.4. MENOPAUSE, HORMONE REPLACEMENT THERAPY AND CARDIOMETABOLIC RISK
8.4.1. WINDOW OF OPPORTUNITY
8.4.2. WOMAN'S AGE
8.4.3. CARDIOMETABOLIC RISK ASSESSMENT 8.4.4.
8.4.4. ROUTE OF ADMINISTRATION
8.4.5. THS WITH ESTROGENS VS. COMBINATION OF ESTROGENS AND PROGESTOGENS
8.5. LIFESTYLE TIPS FOR CARDIOMETABOLIC RISK IN MENOPAUSE
8.5.1. EVALUATION OF THE CHRONOTYPE OF EACH WOMAN
8.5.2. COMBINATION OF AEROBIC, STRENGTH AND PELVIC FLOOR EXERCISE
8.5.3. SUPPLEMENTS, MICRONUTRIENTS, AND BENEFICIAL SUBSTANCES IN MENOPAUSE

1. INTRODUCTION
2. CYTOLYTIC LIVER DISEASES
2.1. VIRAL HEPATITIS
2.1.1. NUTRITIONAL CONSIDERATIONS IN VIRAL HEPATITIS
2.2. NON-ALCOHOLIC STEATOHEPATITIS (FATTY LIVER)
2.2.1. NUTRITIONAL CONSIDERATIONS IN NON-ALCOHOLIC FATTY LIVER DISEASE
2.3. ALCOHOLIC FATTY LIVER
3. CHOLESTATIC LIVER DISEASES
3.1. NUTRITIONAL CONSIDERATIONS IN CHOLESTASIS
3.2. GILBERT'S SYNDROME
4. CHRONIC LIVER FAILURE: LIVER CIRRHOSIS
4.1. NUTRITIONAL CONSIDERATIONS IN SEVERE LIVER FAILURE (CIRRHOSIS)
5. LIVER AND GALLBLADDER CLEANSING

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SUN. 12 ABR 2026 4:00 PM to 8:00 PM
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|
Dr.Álvaro Campillo
|
1. INTRODUCTION
2. CYTOLYTIC LIVER DISEASES
2.1. VIRAL HEPATITIS
2.1.1. NUTRITIONAL CONSIDERATIONS IN VIRAL HEPATITIS
2.2. NON-ALCOHOLIC STEATOHEPATITIS (FATTY LIVER)
2.2.1. NUTRITIONAL CONSIDERATIONS IN NON-ALCOHOLIC FATTY LIVER DISEASE
2.3. ALCOHOLIC FATTY LIVER
3. CHOLESTATIC LIVER DISEASES
3.1. NUTRITIONAL CONSIDERATIONS IN CHOLESTASIS
3.2. GILBERT'S SYNDROME
4. CHRONIC LIVER FAILURE: LIVER CIRRHOSIS
4.1. NUTRITIONAL CONSIDERATIONS IN SEVERE LIVER FAILURE (CIRRHOSIS)
5. LIVER AND GALLBLADDER CLEANSING


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SUN. 26 ABR 2026 4:00 PM to 8:00 PM
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|
María Fraile
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1. CLINICAL CASES OF AUTOIMMUNE DISEASES AND COMORBIDITIES
1.1. RHEUMATOID ARTHRITIS
1.2. THYROID AUTOIMMUNITY
1.3. PSORIASIS
1.4. SJÖGREN'S SYNDROME
2. CLINICAL CASES OF POLYAUTOIMMUNITY AND COMORBIDITIES




1. CANCER IN FIGURES
2. WHAT IS CANCER AND HOW DOES IT DEVELOP?
3. EPIDEMIOLOGY OF CANCER AND WHAT AUTOPSY STUDIES TEACH US ABOUT CANCER
3.1 A HISTORY OF CANCER THAT NO ONE LIKES
3.2 STUNNED BY STATISTICS
3.3 SILENT TESTS AND THE "ANXIOLYTIC" EFFECT OF SCREENING
3.4 WHAT DOES MEDICINE CURRENTLY SAY ABOUT SCREENING PROGRAMS?
4. NUTRITIONAL CONSIDERATIONS IN CANCER PREVENTION AND TREATMENT
4.1 BE ACTIVE. THE IMPORTANCE OF PHYSICAL EXERCISE IN CANCER PREVENTION AND TREATMENT
4.1.1. EFFECTS OF EXERCISE ON HORMONES
4.1.2. EFFECTS OF EXERCISE ON BODY FAT
4.1.3. EFFECTS OF EXERCISE ON INSULIN AND GLUCOSE METABOLISM
4.1.4. REDUCTION OF CHRONIC INFLAMMATION
4.1.5. EFFECTS OF EXERCISE ON THE IMMUNE SYSTEM
4.2 HEALTHY DIET
4.2.1. FOODS RICH IN OMEGA-3
4.2.2. SPECIFIC PROPERTIES OF ALGAE
4.2.3. PROPERTIES OF MUSHROOMS
4.2.4. SPECIFIC PROPERTIES OF CABBAGE AND BROCCOLI
4.2.5. SPECIFIC PROPERTIES OF GARLIC, LEEK, ONION, AND CHIVES
4.2.6. SPECIFIC PROPERTIES OF TOMATOES
4.2.7. SPECIFIC PROPERTIES OF SPICES
4.2.8. HOW TO PREPARE AND DRINK GREEN TEA
4.2.9. TURMERIC, CURCUMIN, AND CURRY
4.2.10. EFFECTS OF FASTING BEFORE CHEMOTHERAPY
5. CACHECHEIA IN CANCER PATIENTS AND HOW TO PREVENT IT. THE PARADOX OF MALNUTRITION IN CANCER PATIENTS

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SUN. 10 MAY 2026 4:00 PM to 8:00 PM
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Dr. Álvaro Campillo
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1. CANCER IN FIGURES
2. WHAT IS CANCER AND HOW DOES IT DEVELOP?
3. EPIDEMIOLOGY OF CANCER AND WHAT AUTOPSY STUDIES TEACH US ABOUT CANCER
3.1 A HISTORY OF CANCER THAT NO ONE LIKES
3.2 STUNNED BY STATISTICS
3.3 SILENT TESTS AND THE "ANXIOLYTIC" EFFECT OF SCREENING
3.4 WHAT DOES MEDICINE CURRENTLY SAY ABOUT SCREENING PROGRAMS?
4. NUTRITIONAL CONSIDERATIONS IN CANCER PREVENTION AND TREATMENT
4.1 BE ACTIVE. THE IMPORTANCE OF PHYSICAL EXERCISE IN CANCER PREVENTION AND TREATMENT
4.1.1. EFFECTS OF EXERCISE ON HORMONES
4.1.2. EFFECTS OF EXERCISE ON BODY FAT
4.1.3. EFFECTS OF EXERCISE ON INSULIN AND GLUCOSE METABOLISM
4.1.4. REDUCTION OF CHRONIC INFLAMMATION
4.1.5. EFFECTS OF EXERCISE ON THE IMMUNE SYSTEM
4.2 HEALTHY DIET
4.2.1. FOODS RICH IN OMEGA-3
4.2.2. SPECIFIC PROPERTIES OF ALGAE
4.2.3. PROPERTIES OF MUSHROOMS
4.2.4. SPECIFIC PROPERTIES OF CABBAGE AND BROCCOLI
4.2.5. SPECIFIC PROPERTIES OF GARLIC, LEEK, ONION, AND CHIVES
4.2.6. SPECIFIC PROPERTIES OF TOMATOES
4.2.7. SPECIFIC PROPERTIES OF SPICES
4.2.8. HOW TO PREPARE AND DRINK GREEN TEA
4.2.9. TURMERIC, CURCUMIN, AND CURRY
4.2.10. EFFECTS OF FASTING BEFORE CHEMOTHERAPY
5. CACHECHEIA IN CANCER PATIENTS AND HOW TO PREVENT IT. THE PARADOX OF MALNUTRITION IN CANCER PATIENTS


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SUN. 24 MAY 2026 4:00 PM to 8:00 PM
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Dr.Álvaro Campillo
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1. INTRODUCTION TO IMMUNOTHERAPY
2. IMMUNOTHERAPY
2.1. CHECKPOINT INHIBITORS
2.2. RESISTANCE AND EVASION MECHANISMS
2.3. CART AND CRISPR
2.4. VACCINES AND ONCOVIRUSES
2.5. CANCER AND METABOLISM

1. WHAT IS CKD AND HOW CAN IT BE DIAGNOSED?
1.1. SECONDARY OR INDIRECT NEGATIVE EFFECTS OF METABOLIC ACIDOSIS
1.2. FLUID-ELECTROLYTE AND ACID-BASE IMBALANCE
1.3. ACCUMULATION OF TOXIC SUBSTANCES
1.4. HORMONAL ALTERATIONS
1.5. SERUM CREATININE VS. GLOMERULAR FILTRATION RATE
2. NUTRITIONAL CONSIDERATIONS IN THE PATIENT WITH CKD
2.1. RECOMMENDATIONS WITH MORE OR LESS SOLID SCIENTIFIC EVIDENCE
2.1.1. HYDRATION IN CKD NOT UNDERGOING DIALYSIS
2.1.2. HYDRATION IN CKD UNDERGOING DIALYSIS
2.1.3. SODIUM INTAKE
2.1.4. POTASSIUM INTAKE
2.1.5. CALCIUM INTAKE
2.1.6. FAT INTAKE
2.1.7. CARBOHYDRATE INTAKE
2.1.8. PROTEIN INTAKE
2.1.9. PHOSPHORUS INTAKE
2.1.10. SCIENTIFIC EVIDENCE REGARDING TYPES OF DIETS RECOMMENDED IN CKD
3. RENAL-INTESTINAL AXIS
3.1. IMPAIRED DIGESTION AND ABSORPTION OF PROTEINS AT THE INTESTINAL LEVEL
3.2. CKD AND INTESTINAL BACTERIA PRODUCING UREASE AND URICASE
3.3. METABOLIC SYNDROME, CKD, AND CARDIOVASCULAR COMPLICATIONS

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SUN. 7 JUN 2026 4:00 PM to 8:00 PM
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Dra.Isabel Barceló
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1. WHAT IS CKD AND HOW CAN IT BE DIAGNOSED?
1.1. SECONDARY OR INDIRECT NEGATIVE EFFECTS OF METABOLIC ACIDOSIS
1.2. FLUID-ELECTROLYTE AND ACID-BASE IMBALANCE
1.3. ACCUMULATION OF TOXIC SUBSTANCES
1.4. HORMONAL ALTERATIONS
1.5. SERUM CREATININE VS. GLOMERULAR FILTRATION RATE
2. NUTRITIONAL CONSIDERATIONS IN THE PATIENT WITH CKD
2.1. RECOMMENDATIONS WITH MORE OR LESS SOLID SCIENTIFIC EVIDENCE
2.1.1. HYDRATION IN CKD NOT UNDERGOING DIALYSIS
2.1.2. HYDRATION IN CKD UNDERGOING DIALYSIS
2.1.3. SODIUM INTAKE
2.1.4. POTASSIUM INTAKE
2.1.5. CALCIUM INTAKE
2.1.6. FAT INTAKE
2.1.7. CARBOHYDRATE INTAKE
2.1.8. PROTEIN INTAKE
2.1.9. PHOSPHORUS INTAKE
2.1.10. SCIENTIFIC EVIDENCE REGARDING TYPES OF DIETS RECOMMENDED IN CKD
3. RENAL-INTESTINAL AXIS
3.1. IMPAIRED DIGESTION AND ABSORPTION OF PROTEINS AT THE INTESTINAL LEVEL
3.2. CKD AND INTESTINAL BACTERIA PRODUCING UREASE AND URICASE
3.3. METABOLIC SYNDROME, CKD, AND CARDIOVASCULAR COMPLICATIONS

1. INTRODUCTION
2. NEPHROLITHIASIS AND NEPHRITIC COLIC
2.1. ACUTE TREATMENT
2.2. NUTRITIONAL AND LIFESTYLE MEASURES TO PREVENT KIDNEY STONES AND KIDNEY COLIC
3. HEMATURIA
4. BLADDER CANCER
5. URINARY INCONTINENCE IN WOMEN (UI)
5.1. TYPES OF UI
5.2. INITIAL QUESTIONS FOR A WOMAN WITH UI
5.3. LIFESTYLE RECOMMENDATIONS AND MEDICAL MANAGEMENT IN WOMEN WITH UI
6. URINARY INFECTIONS AND WOMEN
6.1. IMPORTANT CONCEPTS IN ITUS
6.2. THINGS TO TAKE HOME ABOUT LIFESTYLE IN GENITOURINARY INFECTIONS
6.2.1. DIETARY CHANGES
6.2.2. PHYTOTHERAPY
6.2.3. PROBIOTICS
7. OBESITY AND MEN'S HEALTH
8. DIET AND FERTILITY IN MEN
9. RENAL-INTESTINAL AXIS
9.1. WATER-ELECTROLYTE AND ACID-BASE IMBALANCE
9.1.1. ACID-BASE BALANCE
9.1.2. CALCIUM-PHOSPHORUS METABOLISM
9.2. ACCUMULATION OF TOXIC SUBSTANCES
9.3. HORMONAL ALTERATIONS
9.4. THINGS TO TAKE HOME ABOUT LIFESTYLE, MICROBIOTA, AND KIDNEY DISEASE
9.4.1. HYDRATION
9.4.2. SODIUM INTAKE
9.4.3. POTASSIUM INTAKE
9.4.4. CALCIUM INTAKE
9.4.5. FAT INTAKE
9.4.6. CARBOHYDRATE INTAKE
9.4.7. PROTEIN INTAKE
9.4.8. PHOSPHORUS INTAKE
9.4.9. ALTERATION OF PROTEIN DIGESTION AND ABSORPTION IN THE INTESTINE
9.4.10. CKD AND INTESTINAL BACTERIA PRODUCING UREASE AND URICASE
9.4.11. METABOLIC SYNDROME, CKD AND CARDIOVASCULAR COMPLICATIONS

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SUN. 21 JUN 2026 4:00 PM to 8:00 PM
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Dr.Álvaro Campillo
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1. INTRODUCTION
2. NEPHROLITHIASIS AND NEPHRITIC COLIC
2.1. ACUTE TREATMENT
2.2. NUTRITIONAL AND LIFESTYLE MEASURES TO PREVENT KIDNEY STONES AND KIDNEY COLIC
3. HEMATURIA
4. BLADDER CANCER
5. URINARY INCONTINENCE IN WOMEN (UI)
5.1. TYPES OF UI
5.2. INITIAL QUESTIONS FOR A WOMAN WITH UI
5.3. LIFESTYLE RECOMMENDATIONS AND MEDICAL MANAGEMENT IN WOMEN WITH UI
6. URINARY INFECTIONS AND WOMEN
6.1. IMPORTANT CONCEPTS IN ITUS
6.2. THINGS TO TAKE HOME ABOUT LIFESTYLE IN GENITOURINARY INFECTIONS
6.2.1. DIETARY CHANGES
6.2.2. PHYTOTHERAPY
6.2.3. PROBIOTICS
7. OBESITY AND MEN'S HEALTH
8. DIET AND FERTILITY IN MEN
9. RENAL-INTESTINAL AXIS
9.1. WATER-ELECTROLYTE AND ACID-BASE IMBALANCE
9.1.1. ACID-BASE BALANCE
9.1.2. CALCIUM-PHOSPHORUS METABOLISM
9.2. ACCUMULATION OF TOXIC SUBSTANCES
9.3. HORMONAL ALTERATIONS
9.4. THINGS TO TAKE HOME ABOUT LIFESTYLE, MICROBIOTA, AND KIDNEY DISEASE
9.4.1. HYDRATION
9.4.2. SODIUM INTAKE
9.4.3. POTASSIUM INTAKE
9.4.4. CALCIUM INTAKE
9.4.5. FAT INTAKE
9.4.6. CARBOHYDRATE INTAKE
9.4.7. PROTEIN INTAKE
9.4.8. PHOSPHORUS INTAKE
9.4.9. ALTERATION OF PROTEIN DIGESTION AND ABSORPTION IN THE INTESTINE
9.4.10. CKD AND INTESTINAL BACTERIA PRODUCING UREASE AND URICASE
9.4.11. METABOLIC SYNDROME, CKD AND CARDIOVASCULAR COMPLICATIONS

1. NUTRITION, IMMUNE SYSTEM, AND LENGTH OF HOSPITAL STAY
2. VALIDATION OF HOSPITAL MENUS
3. ENTERAL AND PARENTERAL NUTRITION
4. DRUG-FOOD-SUPPLEMENT INTERACTIONS
4.1. DRUGS: ON AN EMPTY STOMACH OR WITH FOOD?
4.2. PHARMACOLOGICAL INTERACTIONS WITH FRUIT JUICES
4.3. SYNTHETIC LEVOTHYROXINE: EUTIROX ®, DEXNÓN ®, ETC.
4.4. ORAL ANTICOAGULANTS: VITAMIN K ANTAGONISTS: WARFARIN, ACENOCUMAROL SINTROM®
4.5. SOME INTERACTIONS BETWEEN MEDICINAL PLANTS AND SUPPLEMENTS WITH COMMONLY USED DRUGS
4.6. CAN SOME INTERACTIONS BE USED FOR THERAPEUTIC PURPOSES?
5. EXCIPIENTS
6. SUPPLEMENTATION IN HOSPITALIZED PATIENTS
6.1. VITAMIN D AND DRUGS
7. FINAL REFLECTIONS
8. APPENDIX: PHYTOTHERAPY
8.1. MAHÓN CHAMOMILE
8.2. LICORICE
8.3. CLOVE
8.4. CEYLON CINNAMON
8.5. INDIAN TURMERIC
8.6. MINT
8.7. FLAX
8.8. STAR ANISE
8.9. DANDELION
8.10. GINGER
8.11. FENNEL
8.12. BEARBERRY
8.13. LAPACHO
8.14. CHASTE TREE (VITEX AGNUS-CASTUS L.)
8.15. HORSETAIL
8.16. APPLE TREE
8.17. MILK THISTLE
8.18. THISTLE

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SUN. 5 JUL 2026 4:00 PM to 8:00 PM
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María Fraile
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1. NUTRITION, IMMUNE SYSTEM, AND LENGTH OF HOSPITAL STAY
2. VALIDATION OF HOSPITAL MENUS
3. ENTERAL AND PARENTERAL NUTRITION
4. DRUG-FOOD-SUPPLEMENT INTERACTIONS
4.1. DRUGS: ON AN EMPTY STOMACH OR WITH FOOD?
4.2. PHARMACOLOGICAL INTERACTIONS WITH FRUIT JUICES
4.3. SYNTHETIC LEVOTHYROXINE: EUTIROX ®, DEXNÓN ®, ETC.
4.4. ORAL ANTICOAGULANTS: VITAMIN K ANTAGONISTS: WARFARIN, ACENOCUMAROL SINTROM®
4.5. SOME INTERACTIONS BETWEEN MEDICINAL PLANTS AND SUPPLEMENTS WITH COMMONLY USED DRUGS
4.6. CAN SOME INTERACTIONS BE USED FOR THERAPEUTIC PURPOSES?
5. EXCIPIENTS
6. SUPPLEMENTATION IN HOSPITALIZED PATIENTS
6.1. VITAMIN D AND DRUGS
7. FINAL REFLECTIONS
8. APPENDIX: PHYTOTHERAPY
8.1. MAHÓN CHAMOMILE
8.2. LICORICE
8.3. CLOVE
8.4. CEYLON CINNAMON
8.5. INDIAN TURMERIC
8.6. MINT
8.7. FLAX
8.8. STAR ANISE
8.9. DANDELION
8.10. GINGER
8.11. FENNEL
8.12. BEARBERRY
8.13. LAPACHO
8.14. CHASTE TREE (VITEX AGNUS-CASTUS L.)
8.15. HORSETAIL
8.16. APPLE TREE
8.17. MILK THISTLE
8.18. THISTLE

1. INTRODUCTION
1.1. RISK FACTORS FOR POST-SURGICAL COMPLICATIONS
2. THE R.I.C.A. PATHWAY (ENHANCED RECOVERY IN ADULT SURGERY)
3. RECOMMENDATIONS OF THE R.I.C.A. PATHWAY REGARDING NUTRITION AND LIFESTYLE
3.1. PREOPERATIVE NUTRITIONAL ASSESSMENT
3.2. PREOPERATIVE NUTRITIONAL INTERVENTION
3.3. IMMUNONUTRITION
3.4. ASSESSMENT AND TREATMENT OF ANEMIA
3.5. DELAY OR SUSPENSION OF SURGERY IN ANEMIC PATIENTS
3.6. HEMOGLOBIN LEVEL DETERMINATION WITH SUFFICIENT TIME MARGIN
3.7. HEMOGLOBIN LEVEL
3.8. SCREENING FOR IRON DEFICIENCY
3.9. TREATMENT OF PREOPERATIVE ANEMIA
3.10. TREATMENT WITH ORAL IRON
3.11. TREATMENT WITH INTRAVENOUS IRON
3.12. TREATMENT WITH ERYTHROPOIETIC AGENTS
3.13. PREOPERATIVE FASTING
3.14. TREATMENT WITH CARBOHYDRATE DRINKS
3.15. SPECIAL CASES IN PATIENTS WITH DIABETES AND OBESITY
3.16. PERIOPERATIVE BLOOD GLUCOSE MONITORING
3.17. PERIOPERATIVE HYPERGLYCEMIA
3.18. IMMUNONUTRITION (POSTOPERATIVE)
3.19. EARLY POSTOPERATIVE FEEDING
3.20. RESPIRATORY PHYSIOTHERAPY
3.21. MANAGEMENT OF POSTOPERATIVE ANEMIA

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SUN. 19 JUL 2026 4:00 PM to 8:00 PM
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Dr.Álvaro Campillo
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1. INTRODUCTION
1.1. RISK FACTORS FOR POST-SURGICAL COMPLICATIONS
2. THE R.I.C.A. PATHWAY (ENHANCED RECOVERY IN ADULT SURGERY)
3. RECOMMENDATIONS OF THE R.I.C.A. PATHWAY REGARDING NUTRITION AND LIFESTYLE
3.1. PREOPERATIVE NUTRITIONAL ASSESSMENT
3.2. PREOPERATIVE NUTRITIONAL INTERVENTION
3.3. IMMUNONUTRITION
3.4. ASSESSMENT AND TREATMENT OF ANEMIA
3.5. DELAY OR SUSPENSION OF SURGERY IN ANEMIC PATIENTS
3.6. HEMOGLOBIN LEVEL DETERMINATION WITH SUFFICIENT TIME MARGIN
3.7. HEMOGLOBIN LEVEL
3.8. SCREENING FOR IRON DEFICIENCY
3.9. TREATMENT OF PREOPERATIVE ANEMIA
3.10. TREATMENT WITH ORAL IRON
3.11. TREATMENT WITH INTRAVENOUS IRON
3.12. TREATMENT WITH ERYTHROPOIETIC AGENTS
3.13. PREOPERATIVE FASTING
3.14. TREATMENT WITH CARBOHYDRATE DRINKS
3.15. SPECIAL CASES IN PATIENTS WITH DIABETES AND OBESITY
3.16. PERIOPERATIVE BLOOD GLUCOSE MONITORING
3.17. PERIOPERATIVE HYPERGLYCEMIA
3.18. IMMUNONUTRITION (POSTOPERATIVE)
3.19. EARLY POSTOPERATIVE FEEDING
3.20. RESPIRATORY PHYSIOTHERAPY
3.21. MANAGEMENT OF POSTOPERATIVE ANEMIA

1. DRUG-FOOD-SUPPLEMENT-DIAGNOSTIC TEST INTERACTIONS
2. CLINICAL CASES IN HOSPITAL FOOD SERVICE
3. EPILEPSY. CLINICAL CASES OF EPILEPSY
4. CLINICAL CASES OF MULTIPLE SCLEROSIS AND NEUROMYELITIS OPTICA
5. CLINICAL CASES OF BARIATRIC SURGERY AND COMORBIDITIES

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SUN. 2 AGO 2026 4:00 PM to 8:00 PM
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María Fraile
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1. DRUG-FOOD-SUPPLEMENT-DIAGNOSTIC TEST INTERACTIONS
2. CLINICAL CASES IN HOSPITAL FOOD SERVICE
3. EPILEPSY. CLINICAL CASES OF EPILEPSY
4. CLINICAL CASES OF MULTIPLE SCLEROSIS AND NEUROMYELITIS OPTICA
5. CLINICAL CASES OF BARIATRIC SURGERY AND COMORBIDITIES

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SEPTEMBER - DECEMBER 2026
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The Master's Final Project at ICNS is designed to provide the student with genuine scientific competence. For this reason, the module includes several classes on searching for and interpreting scientific articles, giving the student real skills to search for and address clinical practice questions using the available scientific evidence. Additionally, a guided project is carried out, with different submissions that progressively develop the work in parts. The Master?s Final Project therefore follows a logical learning process throughout the different classes and assignments. The classes begin with the most basic concepts, so no prior knowledge is required. The project is developed in the form of continuous assessment over approximately 3 months.
- To be eligible for the Master's Final Project, students must have passed the 3 Specialist Diploma courses and hold a university degree listed in the admission requirements.
- students must formally request access to the Master's Final Project in order to qualify for the Master's Degree, (90 ECTS), which requires an additional payment of 495€.
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Alfonso Bordallo
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· Introduction to PubMed
· Search terms
· Search criteria and filters
· Clinical trials
· Systematic reviews and meta-analyses
· Narrative reviews
· Boolean operators and search strings
· Citation and export
- Evaluation: the first work to be done by the student consists of carrying out different searches of scientific articles, and answering different questions on various issues. The student is corrected and given feedback.
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Alfonso Bordallo
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· Objectives of narrative reviews
· Objectives of clinical trials
· Objectives of systematic reviews and meta-analyses
· Guided reading of a clinical trial
· Key points of a clinical trial
· Forest plot and meta-analysis
· Key points for interpreting a meta-analysis
- Evaluation: in the second assignment, the student chooses a research topic from those proposed by the professors to focus on. Several exercises must be submitted, including conducting a search for relevant articles for the chosen research, presenting key points of physiology and pathophysiology, and describing key aspects of some relevant clinical trials, among others. The work is reviewed, and a video tutoring session is held to provide guidance to the student.
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Alfonso Bordallo
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· Structure of the final paper
· Abstract
· Introduction (physiology, pathophysiology, therapeutic mechanisms)
· Discussion (clinical trials, systematic reviews and meta-analyses, clinical application)
· Conclusions
· Searches conducted
· References
- Evaluation: the third assignment is the final submission. It must expand on what has been developed in the previous submission and integrate it into a single document organized into the proposed sections.
Finally, the student must present and defend their work before the professors on the platform through videoconference. The professors may ask questions about the work.
The student must comply with the specific requirements and regulations of each edition, which will be provided to them. Completing this module requires a minimum level of dedication and effort. If the assignments required for each module are not submitted, the evaluation will be failed and the student will not be able to move on to the next module.
Track Master's in Clinical Nutrition and Endocrinology
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24 ECTS
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25 ECTS
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23 ECTS
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18 ECTS
SEPTEMBER - DECEMBER 2026
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Students who do not wish to do the Master's Final Project or who do not pass the 3 Specialist Diplomas do not have to make this payment and can apply for the approved Specialist Diplomas..
The degrees are exclusive; if you receive the Master's degree you cannot receive the Specialist Diplomas and vice versa.
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24 ECTS
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25 ECTS
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23 ECTS
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18 ECTS
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SEPTEMBER - DECEMBER 2026
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Students who do not wish to do the Master's Final Project or who do not pass the 3 Specialist Diplomas do not have to make this payment and can apply for the approved Specialist Diplomas..
The degrees are exclusive; if you receive the Master's degree you cannot receive the Specialist Diplomas and vice versa.
Objectives of the Master's Program
- To provide the student with a solid knowledge of the pathophysiological level in different pathologies.
- To provide the student with a deep knowledge of clinical nutrition in different pathologies.
- To study whether scientific evidence is compatible with recommendations, guidelines and protocols.
- To interpret analytical tests, in particular those related to nutritional therapy.
- Study different dietary strategies adapted to different preferences.
- Discuss real case studies of the professors' consultations.
- To provide the student with a high knowledge of a wide range of metabolic, endocrine, cardiovascular, hepatic, renal, autoimmune, etc. pathologies.
- To provide the student with consultation skills.
- To provide the student with practical tools.
- To train the student to manage patients with diabetes, endocrine pathologies (thyroid, adrenal, gonads, growth hormone secretion, prolactin secretion disorders, etc.), renal, hepatic, cardiovascular, autoimmune, cancer, etc.
- Introduce the student in principles of clinical hospital nutrition.
- Familiarize the student with real cases and clinical sessions.
- Familiarize the student with relevant scientific evidence.
Access requirements
Higher Technicians in Dietetics (TSD) can study the 3 experts obtaining the following diplomas with ECTS credits:
- Higher University Course in Clinical Nutrition and Health (24 ECTS).
- Higher University Course in Clinical Nutrition and Endocrinology (25 ECTS)
- Higher University Course in Clinical and Hospital Nutrition (23 ECTS)
Online Format
- The Master's program is delivered entirely online through live virtual classes.
- ICNS virtual classes offer more interaction with professors and classmates than a traditional in-person class.
- All classes are recorded and available for a limited time for review.
- Study materials: the course includes several printed books that are shipped to your home.
- In the virtual classroom, students can ask professors questions and interact with their peers.
- All activities, tests, and exams are completed through the virtual platform.
University-Specific Master's Degree

University-Specific Masters Degree ICNS-UCAM
Validity: both permanent training master's degrees and regulated master's degrees are eligible for scoring in public administration competitive exams and selection processes. The scoring of the training depends on the specific rules of each public process or employment pool.
Proprietary master's degrees have been renamed as permanent training master's degrees following the implementation of Royal Decree 822/2021.
Permanent training master's degrees and master's degrees leading to a PhD have different purposes, and both have advantages and disadvantages. The ICNS-UCAM permanent training master's degrees have 60-90 ECTS credits, are issued by universities, and are eligible for scoring. Their main advantage is having more flexible regulations, which allow the design of curricula more oriented toward clinical practice, a more dynamic methodology, a greater variety of topics covered, and a design more focused on student needs. Permanent training master's degrees allow students to be trained in competencies aimed at clinical practice that are often not sufficiently addressed in regulated education.
Thanks to this, there is a greater variety of permanent training degrees that make it possible to offer more specific and comprehensive curricula. Master's degrees leading to a PhD have more structured programs, which limits flexibility in their design. For this reason, universities offer both types of programs to address different needs - whether the goal is to pursue a doctoral and research program in a specific area, or to expand professional competencies, in our case more oriented toward clinical practice.
Therefore, no master's degree is intrinsically better than another; rather, the choice depends on the competencies the student needs, the quality of the institution offering the program, the methodology, the curriculum, the level of updating, the clinical orientation of the subjects, the faculty, and the quality-price ratio.
Student reviews
The material provided is excellent—study materials, the virtual classroom, and, of course, their outstanding nutrition software.
Another thing I love about ICNS is that the professors have a great sense of humor, and you enjoy the classes :)
The professors are great professionals and educators, combining the best of both worlds: great, very interactive classes, clear messages, and the clinical experience and broad perspective of those who see patients.
For that reason, I took this master's program, as years ago I had taken a course with ICNS that I loved, and I wanted to study with them for a longer time. This master's seemed ideal to me because of the complete syllabus and the professors who teach it, as I greatly admire them professionally.
Something that several classmates and I agree on is that ICNS, although all the information is backed by scientific evidence, differs from other places because they actually teach knowledge; they don’t just read one study after another or recite from a book.
Each class is masterful, and you learn a lot. I enjoyed doing the practical exercises for creating diets and have improved my way of working in consultations.
I also want to thank Desirée and Belén for their attention and kindness. They are always there for any problem you have and respond immediately.
Patients put their health in our hands, and we must be accountable. "With great power comes great responsibility" ;)
So, we must train well, beyond collecting degrees. WE HAVE THE POWER TO CHANGE LIVES.
I think I already said this in the previous course, but as a final note I want to repeat my thanks and congratulations for the work you do. At least for me, you are an immeasurable reference. Truly, these courses and classes have greatly changed the way I see both life in general and health in particular. And without even having taken the “Master of Life” yet (the neuroscience one)!
You have all made an effort to keep the topics updated daily, which shows your responsibility as good educators.
You are punctual and answer questions immediately.
Assessment criteria
- Specialist Diploma in Clinical Nutrition and Health: accounting for 24 ECTS on the final grade.
Multiple-choice exams for each module and practical work.
- Maximum number of failures: 1
- Maximum number of absences: 1 unexcused absence or 2 excused absences(*) - Specialist Diploma in Clinical Nutrition and Endocrinology: accounting for 25 ECTS on the final grade.
Multiple-choice exams for each module and practical work.
- Maximum number of failed exams: 1
- Maximum number of absences: 1 unexcused absence or 2 excused absences(*) - Specialist Diploma in Clinical Nutrition and Hospital Nutrition: accounting for 23 ECTS on the final grade.
Multiple-choice exams for each module.
- Maximum number of failures: 1
- Maximum number of absences: 1 unexcused absence or 2 excused absences(*) - Master's Final Project: accounting for 18 ECTS on the final grade
In case of not taking an exam, it will count as 0. The average grade must be at least a 5 to pass.
For the purposes of proper compliance with the regulations and the various academic criteria (completion of activities, deadlines, etc.), only absences due to illness that make it impossible to comply with the program for a medical reason that makes it impossible to complete it on time will be considered justified, as long as it is duly justified (medical leave report or proof of hospital admission).
* For the purposes of class attendance, only shift work in a hospital or health center, as well as sick leave duly justified (medical leave or hospital admission) will be considered as justified absence. Also the time difference in some countries that involve a clear time incompatibility.
Contact Form
Do you have any questions? You can write us here:
c/Madrid, 18
28231 Las Rozas de Madrid
- Clinical Nutrition in Internal And Community Medicine - 60 ECTS
- Clinical Nutrition and Cardiovascular Pathologies - 60 ECTS
- Clinical Nutrition and Endocrinology - 60 ECTS
- Nutrition, Medicine and Hormonal Health in Women - 60 ECTS
- Clinical Nutrition and Digestive Disorders - 60 ECTS
- Sports Nutrition and Body Composition - 60 ECTS
- Clinical Neuroscience - 60 ECTS
- Nutrition and Integrated Clinical Management of Obesity - 60 ECTS
- Eating Disorders - 60 ECTS
- Nutrition And Applied Diet Therapy - 60 ECTS
Calle Madrid, 18
Las Rozas de Madrid 28231, Madrid
[email protected]
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